A catecholamine-secreting tumour

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/catecholamine-secreting-tumour

A course of therapeutic radiation involving the mediastinum

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/course-therapeutic-radiation-involving-mediastinum

A heart transplant

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/heart-transplant

A hypersensitivity reaction of the myocardium to a drug

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/hypersensitivity-reaction-myocardium-drug

A specified autoimmune disease

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/specified-autoimmune-disease

Alcohol consumption

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/alcohol-consumption

Being peripartum

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/being-peripartum

Carbon monoxide poisoning

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/carbon-monoxide-poisoning

Chemotherapeutic agents for cancer

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/chemotherapeutic-agents-cancer

Clinically apparent beriberi

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/clinically-apparent-beriberi

Cocaine or amphetamines

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/cocaine-or-amphetamines

Diabetes mellitus

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/diabetes-mellitus

Envenomation by an animal

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/envenomation-animal

Generalised lipodystrophy

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/generalised-lipodystrophy

Having been a prisoner of war of the Japanese

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/having-been-prisoner-war-japanese

Immunosuppressive drugs following transplantation

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/immunosuppressive-drugs-following-transplantation

Inability to obtain appropriate clinical management for cardiomyopathy

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/inability-obtain-appropriate-clinical-management-cardiomyopathy

Infection with HIV

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/infection-hiv

Infection-related myocarditis

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/infection-related-myocarditis

Infiltration of the myocardium due to a specified disorder

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/infiltration-myocardium-due-specified-disorder

Morbid obesity

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/morbid-obesity

Other specified endocrine disorder

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/other-specified-endocrine-disorder

Selenium deficiency

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/selenium-deficiency

Severe chronic renal failure

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/severe-chronic-renal-failure

Treatment with a specified drug

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-specified-drug

Treatment with an anthracycline

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-anthracycline

Treatment with chloroquine sulphate or chloroquine phosphate or hydroxychloroquine

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-chloroquine-sulphate-or-chloroquine-phosphate-or-hydroxychloroquine

Treatment with corticosteroids

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-corticosteroids

Treatment with lithium or amphotericin B

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-lithium-or-amphotericin-b

Treatment with nonsteroidal anti-inflammatory drugs

Current RMA Instruments

Reasonable Hypothesis SOP	57 of 2024 as amended by 53 of 2025
Balance of Probabilities SOP	58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

ICD-10-AM Codes: I42; I43; O90.3

Brief Description

Cardiomyopathy is a group of acquired diseases of the heart muscle leading to the inability to pump blood effectively and disturbance to the electrical signals controlling heart rate and rhythm.

Confirming the diagnosis

The assessment of this condition is complicated and requires the opinion of a cardiologist.

Investigations may involve echocardiography, electrocardiography and coronary artery angiography/stress testing to exclude other possible diagnoses.

Additional diagnoses covered by SOP

Alcoholic cardiomyopathy
Cytotoxic cardiomyopathy
Dilated cardiomyopathy
Restrictive cardiomyopathy
Familial hypertrophic cardiomyopathy
Idiopathic cardiomyopathy
Infiltrative cardiomyopathy
Primary genetic cardiomyopathy
Radiation cardiomyopathy
Secondary cardiomyopathy
Takotsubo (stress) cardiomyopathy
Myocarditis with persistent mechanical or electrical dysfunction
Hypertrophic cardiomyopathy
Arrhythmogenic cardiomyopathy
Hypertensive cardiomyopathy
Peripartum/Postpartum cardiomyopathy
Septic cardiomyopathy
Left ventricular noncompaction

* This SOP includes genetic cardiomyopathies which do not meet the exclusion criteria

Conditions not covered by SOP

Cardiac transplant rejection #
Myocardial abnormality caused by valvular disease* - Cardiac valve disease SOPs (Rheumatic heart disease; Aortic stenosis; Mitral Valve Prolapse)
Hypertension* - Hypertension SOP
Infective myocarditis* - Myocarditis SOP (May lead to cardiomyopathy as a sequela)
Myocardial abnormality caused by ischaemic heart disease* - Ischaemic heart disease SOP
Pericardial disease including pericarditis* - Pericarditis SOP
Exercise-induced cardiac remodelling (athlete’s heart) #

*another SOP applies

#non-SOP condition

Clinical onset

Once the diagnosis has been established, the clinical date of onset can be taken from the earliest time in which the individual presented with symptoms and signs consistent with cardiomyopathy and that cannot be attributed to other conditions. Common symptoms of cardiomyopathy may include chest pain, shortness of breath, swelling in the lower limb, dizziness, fatigue etc.

Clinical worsening

The prognosis for cardiomyopathy varies depending on the type, severity and underlying cause(s). With treatment, many people can live and function reasonably well. However, the natural history of cardiomyopathy is to slowly progress and worsen. Any considerations related to possible clinical worsening would need the assessment of a cardiologist to ascertain whether there is a true clinical worsening out of keeping with the natural history of the underlying pathology.

Source URL: https://clik.dva.gov.au/ccps-medical-research-library/statements-principles/c-d/cardiomyopathy-g021-4254-425964866748/rulebase-cardiomyopathy/treatment-nonsteroidal-anti-inflammatory-drugs

Cardiomyopathy G021

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

CCPS factors as at 7 March 2007 (G021)

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

A catecholamine-secreting tumour

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

A course of therapeutic radiation involving the mediastinum

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

A heart transplant

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

A hypersensitivity reaction of the myocardium to a drug

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis

Additional diagnoses covered by SOP

Conditions not covered by SOP

Clinical onset

Clinical worsening

A specified autoimmune disease

Current RMA Instruments

57 of 2024 as amended by 53 of 2025

58 of 2024 as amended by 54 of 2025

Changes from previous Intruments

ICD Coding

Brief Description

Confirming the diagnosis